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Past Events > ROS and Mitochondria in Nervous System Function and Disease

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ROS and Mitochondria in Nervous System Function and Disease

27—29 March 2017

Charles Darwin House, London, UK

A Biochemical Society Focused Meeting


Reactive oxygen species (ROS) and mitochondria have key roles in neuronal function and neurological disease. Many outstanding questions remain about the basic mechanisms involving ROS and normal neuronal function and how they contribute to disease. This meeting will provide a cutting edge forum to address these issues by bringing together leading scientists, as well as researchers who are new to the field, to present their current research and discuss future directions.


ROS have long been known to be damaging by-products of metabolism, but the concept that ROS modification of proteins plays a signalling role in the general function of neurons is rapidly gaining ground. Mitochondria generate ATP, but are also important organelles in neuronal signal transduction and are the main source of ROS. This meeting will explore how the roles of ROS and mitochondria intersect in neuronal signal transduction, the underlying biochemical processes and how these mechanisms modulate neuronal function. ROS and mitochondrial signalling are vitally important, not only in nervous system development and function, but also in neurological disease. The meeting will be relevant to researchers interested in basic mechanisms, but also those studying clinical aspects relating to diseases such as Parkinson’s disease, Alzheimer's disease and Amyotropic Lateral Sclerosis among many others.


The major themes of the meeting are:


1.    ROS in signal transduction

2.    Mitochondria as a signalling organelle

3.    ROS and mitochondrial signalling in neuronal function

4.    New techniques for visualising ROS and mitochondria

5.     ROS and mitochondrial signalling in translational medicine


Programme coordinators  Joseph  M. Bateman (King's College London, United Kingdom)

Sean Sweeney (University of York, United Kingdom)


Abstract submission is now closed. 


Registration is now closed.


Oral communication slots are available at this meeting. 

All attendees, particularly researchers in the early stages of their career, are invited to submit a poster abstract for consideration as an oral communication.


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The Biochemical Society offers a programme of grants and bursaries supporting research, attendance at scientific conferences and the sponsorship of events.  


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Topics covered in this meeting have been published in our journals by Portland Press, the wholly-owned trading subsidiary of the Biochemical Society. 


Read this Open Access paper which may be of interest
Understanding and preventing mitochondrial oxidative damage 

Journal: Biochemical Society Transactions

Michael P. Murphy

Date: Oct 19 2016 DOI: 10.1042/BST20160108